Two hours before the press conference was scheduled to begin, we arrived to set up the room and discovered that the hotel had accidentally thrown out all our press kits. All of the information we’d prepared for the press – releases, backgrounders, bios – gone.
Eventually, with minutes to spare, the kits were found and fished from the dumpster out back, a bit worse for wear. We slipped them into new folders, and no one was the wiser.
Except for the one reporter who noticed the coffee grounds stuck to his Cancer Risk Awareness Survey.
In a session devoted to policies that can effect the kind of systemic, real-world changes that can reduce cancer rates, Penn State researcher Barbara J. Rolls, PhD, presented a talk on changing the food environment.
More news on calorie restriction, from Dr. Stephen D. Hursting, a researcher at the University of Texas at Austin. If restricting calories delays and/or prevents tumor formation – as a wide body of research shows – the question is how. And why does obesity increase the risk of cancer? (It does: If you haven’t already read about it – take a look at AICR’s new analysis on the obesity-cancer link.)
Dr. Hursting’s lab is trying to figure out what’s going on in the link between energy intake and cancer. He talks about the animal research involving a key factor in metabolizing energy: IGF-1, which is linked to increased risk of cancer. His research has shown that calorie restriction and obesity both appear to share a common signaling pathway.
He also spoke about some intriguing, relatively new research looking at how exercise plays a role in cancer prevention and energy. Although it looks like exercise does add to the calorie restriction effect, he said, obesity prevention by exercise is not the same as by weight. Two animals can be the same weight – one by diet and the other by exercise – yet there appear to be different signaling effects and gene expressions happening.
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American Institute for Cancer Research
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